MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
MATERIAL SAFETY DATA SHEET
1. CHEMICAL PRODUCT AND COMPANY IDENTIFICATION
CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
Synonyms:
4118A, AN & F CHAMPION庐 ANTIFREEZE
Company Identification
Champion Brands, L.L.C., 1001 Golden Drive, Clinton, MO 64735
PHONE: 800-821-5693 WEBSITE: www.championbrands.com
CAS Registry Number Not Applicable
Synonyms None
Generic/Chemical Name Mixture
Product Type Antifreeze
Preparation Date October 23, 2007
Transportation Emergency Response
CHEMTREC: (800) 424-9300
Product Information
Product Information and MSDS Requests: (800) 821-5693 and www.championbrands.com
2. COMPOSITION/INFORMATION ON INGREDIENTS
COMPONENTS CAS NUMBER AMOUNT
Ethylene Glycol 107-21-1 80.0 - 95.0 % weight
Diethylene glycol 111-46-6 0.0 鈥? 5.0% weight
Dipotassium Phosphate 7758-11-4 1.0 鈥? 2.0% weight
- HARMFUL OR FATAL IF SWALLOWED
- CAUSES EYE IRRITATION
- CONTAINS MATERIAL THAT MAY CAUSE ADVERSE REPRODUCTIVE EFFECTS BASED ON ANIMAL DATA
- POSSIBLE BIRTH DEFECT HAZARD - CONTAINS MATERIAL THAT MAY CAUSE BIRTH DEFECTS BASED ON ANIMAL DATA
- MAY CAUSE DAMAGE TO: KIDNEY
3. HAZARD IDENTIFICATION
NFPA: HEALTH: 1 FLAMMABILITY: 1 REACTIVITY: 0
HMIS: HEALTH: 2 FLAMMABILITY: 1 REACTIVITY: 0
IMMEDIATE HEALTH EFFECTS
Eye: May cause slight transient (temporary) eye irritation. Corneal injury is unlikely. Vapors or mists may cause eye
irritation.
Skin: Prolonged or repeated exposure not likely to cause significant skin irritation. A single prolonged exposure is not
likely to result in the material being absorbed through skin in harmful amounts. Repeated skin exposure may result in
absorption of harmful amounts. Massive contact with damaged skin or of material sufficiently hot to burn skin may
result in absorption of potential lethal amounts.
Ingestion: Single dose oral toxicity is considered to be moderate. Excessive exposure may cause central nervous
system effects, cardiopulmonary effects (metabolic acidosis), and kidney failure. Small amounts swallowed incidental
to normal handling operations are not likely to cause injury; however, swallowing amounts larger than that may cause
serious injury, even death.
Inhalation: At room temperature, exposures to vapors are minimal due to physical properties; higher temperatures
may generate vapor levels sufficient to cause adverse effects.
Systemic (Other Target Organ) Effects: Repeated excessive exposures may cause severe kidney and liver and
gastrointestinal effects. Signs and symptoms of excessive exposure may be central nervous system effects. Signs
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
and symptoms of excessive exposure may be nausea and/or vomiting. Signs and symptoms of excessive exposure
may be anesthetic or narcotic effects. Observations in animals include formation of bladder stones after repeated oral
doses of ethylene glycol. Reports of kidney failure and death in burn patients suggest the ethylene glycol may have
been a factor. The use of topical applications containing this material may not be appropriate in severely burned
patients or individuals with impaired renal function.
Cancer Information: Based on data from long-term animal studies, ethylene glycol is not believed to pose a
carcinogenic risk to man.
Teratology (Birth Defects): Exposure to ethylene glycol has caused birth defects in laboratory animals only at doses
toxic to the mother.
Reproductive Effects: Ethylene glycol has not interfered with reproduction in animal studies except at very high
doses.
DELAYED OR OTHER HEALTH EFFECTS:
Effects of Repeated Overexposure: Repeated inhalation of ethylene glycol mist may produce signs of central
nervous system involvement, particularly dizziness and nystagmus.
Other Effects of Overexposure: repeated skin contact with ethylene glycol may, in a very small proportion of cases,
cause sensitization with the development of allergic contact dermatitis. The incidence is significantly less than 1% with
the undiluted material.
4. FIRST AID MEASURES
Eyes: Immediately flush eyes with large amounts of water for 15 minutes, lifting lower and upper lids. Get medical
attention as soon as possible. Contact lenses should never be worn when working with this chemical.
Skin: Flush area of skin contact immediately with large amounts of water for at least 15 minutes while removing
contaminated clothing. If irritation persists after flushing, get medical attention promptly. Wash clothing before re-use.
Inhalation: If inhaled, immediately remove victim to fresh air and call emergency medical care. If not breathing, give
artificial respiration. If breathing is difficult, give oxygen.
Ingestion: Obtain medical attention immediately. If patient is fully conscious, give two glasses of water. Do not induce
vomiting. If medical advice is delayed, and if the person has swallowed a moderate volume of material (a few ounces),
then give three to four ounces of hard liquor, such as whisky. For children, give proportionally less liquor, according to
weight.
Notes to Physician: It is estimated that the lethal oral dose to adults is of the order of 1.0 ml/kg. Ethylene glycol is
metabolized by alcohol dehydrogenate to various metabolites including glyceraldehydes, glycolic acid and oxalic acid
which cause an elevated anion-gap metabolic acidosis and renal tubular injury. The signs and symptoms in ethylene
glycol poisoning are those of metabolic acidosis, CNS depression, and kidney injury. Urinalysis may show
albuminuria, hematuria and oxaluria. Clinical chemistry may reveal anion-gap metabolic acidosis and uremia. The
currently recommended medical management of ethylene glycol poisoning includes elimination of ethylene glycol and
metabolites, correction of metabolic acidosis and prevention of kidney injury. It is essential to have immediate and
follow up urinalysis and clinical chemistry. There should be particular emphasis on acid-base balance and renal
function tests. A continuous infusion of 5% sodium bicarbonate with frequent monitoring of electrolytes and fluid
balance is used to achieve correction of metabolic acidosis and forced diuresis. As a competitive substrate for alcohol
dehydrogenase, ethanol is antidotal. Given in the early stages of intoxication, it blocks the formulation of nephrotoxic
metabolites. A therapeutically effective blood concentration of ethanol is in the range 100-150 mg/dl, and should be
achieved by a rapid loading dose and maintained by intravenous infusion. For severe and/or deteriorating cases,
hemodialysis may be required. Dialysis should be considered for patients who are symptomatic, have severe
metabolic acidosis, a blood ethylene glycol concentration greater than 25 md/dl, or compromise of renal functions.
A more effective intravenous antidote for physician use is 4-methylpyrazole, a potent inhibitor of alcohol
dehydrogenases, which effectively blocks the formation of toxic metabolites of ethylene glycol. It has been used to
decrease the metabolic consequences of ethylene glycol poisoning before metabolic acidosis coma, seizures, and
renal failure have occurred. A generally recommended protocol is a loading dose of 15 mg/kg followed by 10 mg/kg
every 12 hours for 4 doses and then 15 mg/kg every 12 hours until ethylene glycol concentrations are below 20
mg/100 ml. Slow intravenous infusion is required. Since 4-methyplyrozole is dialyzable, increased dosage may be
necessary during hemodialysis. Additional therapeutic measures may include the administration of cofactors involved
in the metabolism of ethylene glycol. Thiamine (100 mg) and pyridoxine (50 mg) should be given every six hours.eyes
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
with water immediately while holding the eyelids open. Remove contact lenses, if worn, after initial flushing, and
continue flushing for at least 15 minutes. Get medical attention if irritation persists.
Pulmonary edema with hypoxemia has been described in a number of patients following poisoning with ethylene
glycol. The mechanism of production has not been elucidated, but it appears to be non-cardiogenic in origin in several
cases. Respiratory support with mechanical ventilation and positive end expiratory pressure may be required. There
may be cranial nerve involvement in the late stages of toxicity from swallowed ethylene glycol. In particular, effects
have been reported involving the seventh, eighth and ninth cranial nerves, presenting with bilateral facial paralysis,
diminished hearing and dysphasia.
5. FIRE FIGHTING MEASURES
FIRE CLASSIFICATION:
OSHA Classification (29 CFR 1910.1200): Not classified by OSHA as flammable or combustible.
NFPA RATINGS: Health: 1 Flammability: 1 Reactivity: 0
FLAMMABLE PROPERTIES:
Flashpoint: 119掳C (247掳F)
Autoignition Temperature: Autoignition temperature for ethylene glycol is 398掳C (748掳F).
Flammability Limits - % of vapor concentration at which product can ignite in presence of spark.
Lower Flammability Limit: 3.2%
Upper Flammability Limit: 15.3%
Hazardous Combustion Products: Hazardous combustion products may include and are not limited to carbon
monoxide, carbon dioxide and trace amounts of aldehydes and organic acids. When available oxygen is limited, as in
a fire or when heated to very high temperatures by a hot wire or plate, carbon monoxide and other hazardous
compounds such as aldehydes might be generated.
Extinguishing Media: Water fog or fine spray. Alcohol resistant foams (ATC type) are preferred if available. General
purpose synthetic foams (including AFFF) or protein foams may function, but much less effectively. Carbon dioxide.
Dry chemical. Do not use direct water stream. May spread fire.
Fire Fighting Instructions: No fire and explosion hazards expected under normal storage and handling conditions
(i.e. ambient temperatures). However, ethylene glycol or solutions of ethylene glycol and water can form flammable
vapors with air if heated sufficiently. Keep people away. Isolate fire area and deny unnecessary entry.
Protective Equipment for Fire Fighters: Wear positive -pressure, self-contained breathing apparatus (SCBA) and
protective fire fighting clothing (includes fire-fighting helmet, coat, pants, boots and gloves).
6. ACCIDENTAL RELEASE INFORMATION
Protect People: Material is moderately toxic when ingested. Take adequate precautions to keep people, especially
children away from spill site. PVC-coated rubber gloves and monogoggles or face shield can be used during cleanup
of spill site. Product on surfaces can cause slippery conditions. Practice reasonable care and cleanliness. Avoid
breathing spray mists if generated. Keep out of reach of children. Product may become a solid at temperatures below
-18oC (0oF). Do not store near food, foodstuffs, drugs or potable water supplies.
Protect the Environment: Do not dump used product or diluted material into sewers, on the ground, or into any body of
water.
Cleanup: Small spills: Soak up with absorbent material. Large spills: Dike and pump into suitable containers for
disposal. Ensure compliance with all applicable statues that require notification of appropriate government officials.the
source of the release if you can do it without risk. Contain release to prevent further contamination of soil, surface
water or groundwater. Clean up spill as soon as possible, observing precautions in Exposure Controls/Personal
Protection. Use appropriate techniques such as applying non-combustible absorbent materials or pumping. Where
feasible and appropriate, remove contaminated soil. Place contaminated materials in disposable containers and
dispose of in a manner consistent with applicable regulations.
7. HANDLING AND STORAGE
General Handling Information: Do not taste or swallow antifreeze or solution. Keep out of the reach of children and
animals.
General Storage Information: Do not store in open or unlabeled containers. Keep out of reach of children. Product
may become a solid at temperatures below -18掳C (0掳F). Do not store near food, foodstuffs, drugs or potable water
supplies.
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
8. EXPOSURE CONTROL/PERSONAL PROTECTIVE EQUIPMENT
GENERAL CONSIDERATIONS:
Consider the potential hazards of this material (see Section 3), applicable exposure limits, job activities, and other
substances in the work place when designing engineering controls and selecting personal protective equipment. If
engineering controls or work practices are not adequate to prevent exposure to harmful levels of this material, the
personal protective equipment listed below is recommended. The user should read and understand all instructions
and limitations supplied with the equipment since protection is usually provided for a limited time or under certain
circumstances.
ENGINEERING CONTROLS:
Use process enclosures, local exhaust ventilation, or other engineering controls to control airborne levels below the
recommended exposure limits.
PERSONAL PROTECTIVE EQUIPMENT
Respiratory Protection: Respiratory protection is required if airborne concentration exceeds TLV. At any detectable
concentration any self-contained breathing apparatus with a full face piece and operated in a pressure-demand or
other positive pressure mode or any supplied-air respirator with a full face piece and operated in a pressure-demand
or other positive pressure mode in combination with an auxiliary self-contained breathing apparatus operated in
pressure-demand or other positive pressure mode.
Escape: Any air-purifying full face piece respirator (gas mask) with a chin-style or front- or back-mounted organic
vapor canister or any appropriate escape-type self-contained breathing apparatus.
Skin Protection: Protective gloves recommended when prolonged skin contact cannot be avoided. Polyethylene;
Neoprene; Nitrile; Polyvinyl alcohol; Natural Rubber, Butyl Rubber. Safety shower should be available.
Eye Protection: Safety goggles and face shield. Emergency eyewash should be available. Contact lenses should not
be worn when working with this chemical.
Engineering Controls: Use general or local exhaust ventilation to meet TLV requirements.
Occupational Exposure Limits:
Component Exposure Limits Skin Form
Ethylene glycol 100 mg/m3 CEILING ACGIH Aerosol
Ethylene glycol 125 mg/m3 CEILING OSHA -vacated
50 ppm CEILING OSHA 鈥? vacated
100 mg/m3 CEILING UCC Aerosol and Vapor
Diethylene glycol 50 ppm TWA8 AIHA WEEL Aerosol and Vapor
Diethylene glycol 10 mg/m3 TWA8 AIHA WEEL Aerosol
9. PHYSICAL AND CHEMICAL PROPERTIES
Attention: the data below are typical values and do not constitute a specification.
Boiling Range: 171 - 175掳C (339 - 348掳F)
Freeze Point: -18掳C (0掳F)
Specific Gravity (Water =1): 1.12
Pounds/Gallons: 9.3
Vapor Pressure (mm of Hg) @ 20C: <0.1
Vapor Density (air=1): 2.1
Water Solubility: Complete
Evaporation Rate (BuAc = 1): Nil
% Volatile By Volume: 97.0
Appearance: Green
Odor: Mild
pH (50% Water Solution): 10.5-11.0
10. STABILITY AND REACTIVITY
Chemical Stability: This material is considered stable under normal ambient and anticipated storage and handling
conditions of temperature and pressure. Keep away from flame
Incompatibility With Other Materials: Strong acid or oxidizing agents
Hazardous Decomposition Products: Incomplete combustion may produce CO gas
Hazardous Polymerization: Hazardous polymerization will not occur.
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
11. TOXILOGICAL INFORMATION
IMMEDIATE HEALTH EFFECTS
Skin: The dermal LD50 has not been determined.
Ingestion: The lethal dose in humans is estimated to be 100 ml (3 ozs.). The oral LD50 for rats is in the 6000-13,000-
mg/kg range.
Mutagenicity (The Effects on Genetic Material): In vitro mutagenicity studies were negative. Animal mutagenicity
studies were negative.
Significant Data with Possible Relevance to Humans: Ethylene glycol has been shown to produce dose-related
teratogenic effects in rats and mice when given by gavage or in drinking water at high concentrations or doses. The
no-effect doses for developmental toxicity for ethylene glycol given by gavage over the period of organogenesis has
been shown to be 150 mg/kg/day for the mouse and 500 mg/kg/day for the rat. Also, in a preliminary study to asses
the effects of exposure of pregnant rats and made to aerosis at concentrations of 150, 1000 and 25000 mg/m3 for 6
hours a day throughout the period of organogenesis, teratogenic effects were produced at the highest concentration,
but only in mice. The conditions of these latter experiments did not allow a conclusion as to whether the
developmental toxicity was mediated by inhalation of aerosol percutaneous absorption of ethylene glycol from
contaminated skin, or swallowing ethylene glycol as a result of grooming the wetted coat. In a further study,
comparing effects from high aerosol concentration by whole-body or nose-only exposure, it was shown that nose-only
exposure resulted in maternal toxicity (1000 and 25000 mg/m3) and developmental toxicity with minimal evidence of
teratogenicity (2500 mg/m3). The no-effects concentration (based on maternal toxicity) was 500 mg/m3. In a further
study in mice, no teratogenic effects could be produced when ethylene glycol was applied to skin of pregnant mice
over the period of organogenesis. The above observations suggest that ethylene glycol is to be regarded as an animal
teratogen. There is currently no available information to suggest that ethylene glycol has caused birth defects in
humans. Cutaneous application of ethylene glycol is ineffective in producing developmental toxicity. Exposure to high
aerosol concentrations is only minimally effective in producing developmental toxicity. The major route for producing
developmental toxicity is perorally. Two chronic feeding studies, using rats and mice, have not produced any evidence
that ethylene glycol causes dose-related increases in tumor incidence or a different pattern of tumors compared with
untreated controls. The absence of carcinogenic potential for ethylene glycol has been supported by numerous in vitro
genotoxicity studies showing that it does not produce mutagenic or clastogenic effects.
A chronic dietary feeding study of diethylene glycol with rats showed mild kidney injury at 1%, while concentrations of
2% and 4% caused more marked kidney injury. In addition, at 2% and 4% of diethylene glycol in the diet, some rats
developed benign papillary tumors in the urinary bladder. These have been attributed to the presence of urinary
bladder calcium oxalate stones. No evidence for carcinogenicity was found with a chronic skin-painting study with
diethylene glycol in mice. The absence of a direct chemical carcinogenic effect addords with the results in vitro
genotoxicity studies that show that it does not produce mutagenic or clastogenic effects. A feeding study employing
up to 5.0% diethylene glycol in the diet failed to produce any teratogenic effects. In a mouse continuous breeding
study with large doses of diethylene glycol in drinking water, there was evidence for reproductive toxicity at 3.5%
(equivalent to 6.1 g/kg/day) as reduced number of litter, live pups per litter and live pup weight. No such effects were
seen at 1.75% (approximately 3.05 g/kg/day). The relevance of these very high dosages to human health is uncertain.
Pregnant rats receiving undiluted diethylene glycol by gavage over the period of organogenesis had toxic effects at
4.0 and 8.0 ml/kg/day as mortality, decreased body weight, decreased food consumption increased water
consumption and increased liver and kidney weights. Fetotoxicity was seen only at these maternally toxic dosages.
Decreased fetal body weight occurred at 8.0 ml/kg/day, and increased skeletal variants at 4.0 and 8.0 ml/kg/day. No
embryotixic or teratogenic effects were seen. Neither maternal toxicity nor fetotoxicity occurred at 1.0 ml/kg/day. In a
study with mice also receiving undiluted diethylene glycol over the period of organogenesis, maternal toxicity occurred
at 2.5 and 10.0 ml/kg/day, but not at 0.5 ml/kg/day. Definitive developmental toxicity was not seen in this species.
ACUTE TOXICITY
Peroral: The lethal dose in humans is estimated to be 3 oz. or 100 ml.
Rat: LD50 (6000 鈥? 13000) mg/kg
Percutaneous: Rabbit: LD50 = >22270 mg/kg; 24 h occluded
Inhalation: Rat: 8-hour exposure, substantially saturated vapor studies, dynamic generation method
Mortality: 0/6
Inhalation: Mist/vapor study, rat, at 170掳C, 8-hour exposure = 2.2 mg/l
Mortality: 0/6
Inhalation: Rat: 8-hour exposure, fog = 10000 ppm; 65掳 - 70掳C
Mortality: 0/6
IRRITATION
Skin: Rabbit: 24-hour occluded contact, 0.5 ml
Results: Minor erythema and edema
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
Skin: Human: Primary irritation patch test, 48-hour occluded, 0.2 ml
Results: Evidence of irritation
Eye: Rabbit: 0.1 ml
Results: Minor transient iritis, conjunctival irritation with discharge
REPEATED EXPOSURE
In a 7-day dietary study with rats, a significant increase in kidney weights in females was observed at 5.0 gm/kg. The
NOEL was 2.5 gm/kg.
In a 24-month dietary study with rats, increased mortality in males was observed at the highest dose, 1.0 gm/kg/day.
There were multiple signs: mineralization of several organs, including the cardiac vessels, cardiac muscle, vas
deferens, stomach and pulmonary vessels; cellular hyperplasia of the parathyroids, hemosiderosis of the spleen,
myocardial fibrosis, portal fibrosis of the liver, bile duct hyperplasia and hydronephrosis and oxylate nephrosis of the
kidneys. Ethylene glycol was not oncogenic.
In a 90-day dietary study with dogs, repeated exposures to 2.5 gm/kg resulted in acute renal failure and deaths. Th e
NOAEL was 1.0 gm/kg.
SENSITIZATION (ANIMAL AND HUMAN STUDIES)
Repeated skin contact with ethylene glycol may, in a very small proportion of cases, cause sensitization with the
development of allergic contact dermatitis. The incidence is significantly less than 1% with the undiluted material.
REPRODUCTIVE TOXICITY
A three-generation study indicated that ethylene glycol did not affect reproductive parameters at dietary
concentrations up to 1.0 gm/kg/day in any generation.
CHRONIC TOXICITY AND CARCINOGENICITY
Two chronic feeding studies, using rats and mice, have not produced any evidence that ethylene glycol causes dose-
related increases in tumor incidence or a different pattern of tumors compared with untreated controls. The absence
of a carcinogenic potential for ethylene glycol has been supported by numerous in vitro genotoxicity studies showing
that it does not produce mutagenic or clastogenic effects.
GENETIC TOXICOLOGY
In Vitro: Ethylene glycol was devoid of genotoxic activity in an Ames test, forward gene mutation and sister chromatid
exchange (SCE) studies in Chinese Hamster Ovary (CHO) cells and an in vitro cytogenetics study.
In Vivo: Ethylene glycol by three different routes (intravenous, peroral and percutaneous) demonstrates apparent
first-order pharmacokinetic behavior for the disposition in and the elimination from the plasma. Dose-dependent
changes occur for the elimination of metabolites in the urine and as 14CO2 after single doses for the intravenous and
peroral, but not the percutaneous route. The hypothesis from literature sources exists that developmental toxicity is
caused by a metabolite of ethylene glycol, called glycolic acid, and not parent ethylene glycol. Under most conditions
of ethylene glycol exposure, the glycolic acid metabolite is present in the blood in very low levels. However, it can
become the major metabolite following large doses of ethylene glycol due to saturation of glycolic acid oxidation
and/or elimination. When levels of this acidic metabolite exceed the capacity of maternal blood buffers to neutralize it,
a maternal metabolic acidosis ensues, which has been hypothesized to be the true agent responsible for ethylene
glycol induced developmental toxicity. Research suggests that ethylene glycol developmental toxicity is due to a dose-
rate dependent toxicokinetic shift leading to glycolate accumulation and metabolic acidosis.
ADDITIONAL STUDIES
Ethylene glycol has been shown to produce dose-related teratogenic effects in rats and mice when given by gavage
or in drinking water at high concentrations or doses. The no-effect doses for developmental toxicity for ethylene glycol
given by gavage over the period of organogenesis has been shown to be 150 mg/kg/day for the mouse and 500
mg/kg/day for the rat. Also, in a preliminary study to assess the effects of exposure of pregnant rats and mice to
aerosols at concentrations of 150, 1000 and 2500 mg/m3 for 6 hours a day throughout the period of organogenesis,
teratogenic effects were produced at the highest concentration, but only in mice. The conditions of these latter
experiments did not allow a conclusion as to whether the developmental toxicity was mediated by inhalation of
aerosol, percutaneous absorption of ethylene glycol from contaminated skin, or swallowing of ethylene glycol as a
result of grooming the wetted coat. In a further study, comparing effects from high aerosol concentration by whole-
body or nose-only exposure, it was shown that nose-only exposure resulted in maternal toxicity (1000 and 2500
mg/m3) and developmental toxicity with minimal evidence of teratogenicity (2500 mg/m3). The no-effects
concentration (based on maternal toxicity) was 500 mg/m3. In a further study in mice, no teratogenic effects could be
produced when ethylene glycol was applied to the skin of pregnant mice over the period of organogenesis. The above
observations suggest that ethylene glycol is to be regarded as an animal teratogen. There is currently no available
information to suggest that ethylene glycol has caused birth defects in humans. Cutaneous application of ethylene
glycol is ineffective in producing developmental toxicity. Exposure to high aerosol concentrations is only minimally
effective in producing developmental toxicity.
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
12. ECOLOGICAL INFORMATION
ENVIRONMENTAL FATE
Movement & Partitioning: Bioconcentration potential is low (BCF less than 100 or Log Kow less than 3). Log
octanol/water partition coefficient (log Kow) is -1.36. Henry's Law Constant (H) is 6.0E-08 atm-m3/mol.
Bioconcentration factor (BCF) is 10 in golden orfe.
Degradation & Transformation: Biodegradation under aerobic static laboratory conditions is high (BOD20 or
BOD28/ThOD greater than 40%). 5-Day biochemical oxygen demand (BOD5) is 0.78 p/p. 10-Day biochemical oxygen
demand (BOD10) is 1.06 p/p. 20-Day biochemical oxygen demand (BOD20) is 1.15 p/p. Theoretical oxygen demand
(THOD) is calculated to be 1.29 p/p. Biodegradation may occur under both aerobic and anaerobic conditions (in either
the presence or absence of oxygen). Inhibitory concentration (IC50) in OECD "Activated Sludge, Respiration Inhibition
Test" (Guideline # 209) is < 1000 mg/L. Degradation is expected in the atmospheric environment within days to
weeks.
Ecotoxicology: Material is practically non-toxic to aquatic organisms on an acute basis (LC50 greater than 100 mg/L
in most sensitive species). Acute LC50 for fathead minnow (Pimephales promelas) is 51000 mg/L. Acute LC50 for
bluegill (Lepomis macrochirus) is 27549 mg/L. Acute LC50 for rainbow trout (Oncorhynchus mykiss) is about 18000-
46000 mg/L. Acute LC50 for guppy (Poecilia reticulata) is 49300 mg/L. Acute LC50 for water flea (Daphnia magna) is
46300-51100 mg/L. Acute LC50 for the cladoceran Ceriodaphnia dubia is 10000-25800 mg/L. Acute LC50 for crayfish
is 91430 mg/L. Acute LC50 for brine shrimp (Artemia salina) is 20000 mg/L. Acute LC50 for golden orfe (Leuciscus
idus) is greater than 10000 mg/L. Acute LC50 for goldfish (Carassius auratus) is greater than 5000 mg/L. Growth
inhibition EC50 for green alga Selenastrum capricornutum is 9500-13000 mg/L.
BOD (% Oxygen Consumption):
Day 5 Day 10 Day 15 Day 20 Day 30
51% 80% 97%
ECOTOXICITY
Toxicity to Micro-organisms:
Bacterial / NA: 16 h; IC50
Result Value: >10000 mg/l
Toxicity to Aquatic Invertebrates:
Daphnia: 48 h; LC50
Result Value: >100000 mg/l
Toxicity to Fish
Fathead Minnow: 94 h; LC50
Result Value: 70000 mg/l
FURTHER INFORMATION
Chemical Oxygen Demand (COD) 鈥? Measured: 1.29 mg/mg
Theoretical Oxygen Demand (THOD) 鈥? Calculated: 1.30 mg/mg
Octanol/Water Partition Coefficient 鈥? Measured: -1.36
13. DISPOSAL INFORMATION
DO NOT discharge to sewer. Wear appropriate personal protection. Take up with sand, vermiculite, or similar inert
material. Dispose in accordance with federal, state and local regulations.
14. TRANSPORTATION INFORMATION
The description shown may not apply to all shipping situations. Consult 49CFR, or appropriate Dangerous Goods
Regulations, for additional description requirements (e.g., technical name) and mode-specific or quantity-specific
shipping requirements.
U.S. DEPARTMENT OF TRANSPORTATION: Non-Bulk is not regulated by the US D.O.T. (in quantities under 5,000 lbs in
any one inner package)
IATA: Non-Bulk is not regulated by IATA
IMDG: Non-Bulk is not regulated by IMDG (in quantities under 5,000 lbs in any one inner package)
15. REGULATORY INFORMATION
THIS PRODUCT CONTAINS COMPONENT(S) CITED ON THE FOLLOWING REGULATIONS.
Chemical Name Cas Number
Ethylene Glycol 107-21-1
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MSDS: CHAMPION庐 MSDS 2010 SERIES ANTIFREEZE
United States 鈥? TSCA Inventory: Listed
Water Standards: No data available
Atmospheric Standards: Clean Air Act (1990) - List of Hazardous Air Contaminants: listed
CERCLA: Reportable Quantity (RQ): 5,000 pounds (532 gallons)
OSHA Hazard Communication Standard: This product is a 鈥渉azardous chemical鈥? as defined by the OSHA Hazard
Communication Standard, 29 CFR 1910.1200.
SARA Title III: Section 311/312 - Categories: Acute hazard; chronic hazard
Section 312 - Inventory Reporting: Ethylene glycol is subject to Tier I and/or Tier II annual inventory reporting.
Section 313 - Emission Reporting: Ethylene glycol is subject to Form R reporting requirements.
Section 302 - Extremely Hazardous Substances: Ethylene glycol is not listed.
CHEMICAL INVENTORIES:
State Right-To-Know:
Director's List of Hazardous Substances: listed
Florida - Hazardous Substances List: listed
Massachusetts - Right-to-Know List: listed
Minnesota - Haz. Subs. List: listed (particulate and vapor)
New Jersey - Right-to-Know List (Total): Present greater than 1.0%
Pennsylvania Right-to-Know List: environmental hazard
California - Exposure Limits - Ceilings: vapor-50 ppm ceiling; 125 mg/m3 ceiling
California Proposition 65 (Safe Drinking Water and Toxic Enforcement Act of 1986): The normal
consumer use of this product does not result in exposure to chemicals known to the state of California to
cause Cancer and/or reproductive harm above the significant risk level for carcinogens or the maximum
allowable dose levels for reproductive toxins. Warnings are not required for consumer packaging. However,
industrial or other occupational use of this product at higher frequency and using larger quantities of this
product may result in exposures exceeding these levels and are labeled accordingly.
California SCAQMD Rule 443.1 (South Coast Air Quality Management District Rule 443.1, Labeling of
Materials Containing Organic Solvents): VOC: Vapor pressure 0.06 mmHg at 20掳C, 1113.38 g/l
Canadian Regulations: This product has been classified in accordance with the hazard criteria of the Controlled
Products Regulations (CPR) and the MSDS contains all the information required.
WHMIS Information: D2A - material has potential toxic effects. Refer elsewhere in the MSDS for specific warnings and safe
handling information. Refer to the employer's workplace education program.
16. DISCLAIMER
REVISION STATEMENT: Revision updates many sections and the MSDS should be read in its entirety.
ABBREVIATIONS THAT MAY HAVE BEEN USED IN THIS DOCUMENT:
TLV - Threshold Limit Value TWA - Time Weighted Average
STEL - Short-term Exposure Limit PEL - Permissible Exposure Limit
CHA - Champion LLC CAS - Chemical Abstract Service Number
NDA - No Data Available NA - Not Applicable
<= - Less Than or Equal To >= - Greater Than or Equal To
Prepared according to the OSHA Hazard Communication Standard (29 CFR 1910.1200) and the ANSI MSDS
Standard (Z400.1) by Champion LLC, 1001 Golden Drive, Clinton, Missouri 64735.
The information presented herein has been compiled from sources considered to be dependable and is accurate to the best knowledge of
Champion Brands, L.L.C. Champion Brands, L.L.C., makes no warranty whatsoever expressed or implied of merchantability or fitness for the
particular purpose, regarding the accuracy of such data or the results to be obtained from the use thereof. Champion Brands, L.L.C., assumes no
legal responsibility for use or reliance upon this data. Since this information may be applied under conditions beyond our control and with which we
may be unfamiliar and since data made available subsequent to the date hereof may suggest modifications of the information, we do not assume
any responsibility for the results of its use. This information is furnished upon condition that the person receiving it shall make his own determination
of the suitability of the material for his particular purpose.
REV DATE: 10/23/2007 PAGE 8 OF 8
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